In combination with the intestinal damage caused by coccidiosis, excess mucin production as a consequence to the replication of sporozoites within the mucosal epithelium provides a beneficial source of nutrients for C. perfringens proliferation, thus further exacerbating the inflammation and disruption of the gut and negatively impacting digestive and absorptive capacities (Adhikari et al., 2020). The gene discussed is MUC5AC; the disease is coccidiosis.