The results showed that clusters A and C were enriched for canonical cancer signaling pathways and processes, such as the VEGF, mTOR, ERBB, MAPK, Wnt, TGF-β, hedgehog, and Notch pathways, as well as tight junctions, adherens junctions, cell cycle, non-homologous end joining, mitotic sister chromatid cohesion, mRNA processing, and related GO terms compared with samples exhibiting cluster B patterns (Additional file 2: Fig. S5), further indicating the involvement of m6A modification in cervical cancer progression. This evidence concerns the gene TGFB1 and cancer.