To better understand whether and how m6A regulators contribute to cervical cancer progression, we first identified 9 m6A writers (WTAP, ZC3H13, METTL3, METTL14, METTL16, VIRMA, RBM15B, RBM15, and CBLL1), 15 m6A readers (FMR1, hnRNPA2B1, hnRNPC, YTHDF1/2/3, YTHDC1/2, LRPPRC, IGF2BP1, IGF2BP2, IGF2BP3, RBMX, EIF3A, and ELAVL1), and 2 m6A erasers (FTO and ALKBH5) that were previously reported to have pathogenic roles in other human cancer types [7, 8]. This evidence concerns the gene RBMX and cancer.