EGF and psoriasis: It is postulated that thyroid hormones bind to the receptors present on the skin and produce epidermal growth factor (EGF), which leads to keratin synthesis and epidermal hyperplasia [2,3]. Several studies have implicated the role of T-helper type 1 (TH-1) cell-mediated inflammation and their various inflammatory pathways in the immunopathogenesis of psoriasis and AITD [4]. There is no current evidence regarding the association of human leukocyte antigen (HLA).