The top 5 genes were Myl9 (myosin light chain 9), Csrp2 (cysteine and glycine rich protein 2), Acta2 (smooth muscle (SM) actin α 2, SM α-actin), Tagln (transgelin, SM22α), and Rgs5 (Regulator of G protein signaling 5) [8], suggesting the potential importance of these VSMC genes in the development of AAA. The gene discussed is TAGLN; the disease is triple-A syndrome.