MTARC1 and metabolic dysfunction-associated steatotic liver disease: MTARC1 is a molybdenum‐dependent enzyme that reduces N‐oxygenated molecules.[19, 49] We found that alanine‐165 is highly conserved and threonine‐165 may disrupt the alpha‐helix and its ability to bind molybdenum, although precisely how this reduces the severity of NAFLD will require further characterization.