In BCC, the expressions of PDGFR and prolyl-4-hydroxylase mRNA were augmented among the CAF activation markers, leading to increased CAF-related chemokines CCL17, CCL18, CCL22, CCL15, CXCL12, and IL-6 in both the tumor site and peritumoral skin [98]. This evidence concerns the gene CCL17 and neoplasm.