A year later, Narges et al. [28] also confirmed the antitumor activity of miR-424-5p in BC cells and proposed the corresponding mechanism, which is that miR-424-5 can target PD-L1 and regulate the expression of the PTEN/PI3K/AKT/mTOR signaling pathway to inhibit cell proliferation and lock the cell cycle in the G2 phase. This evidence concerns the gene MTOR and breast cancer.