By predicting that SNHG12 has a presumptive binding site related to the seed sequence of miR-424-5p, the experimental verification showed that the tumor-promoting effect of SNHG12 was through acting as a molecular sponge of miR-424-5p, thus negatively regulating the expression of miR-424-5p in cervical cancer to promote the proliferation and invasion of cervical cancer cells. Here, SNHG12 is linked to neoplasm.