PCDH17 and cancer: The similarities in the epigenetic mechanism of regulation between cDLBCL and other human cancers, as well as evidence of antitumourigenic activity of CiDEA, MAL and PCDH17 after ectopic re-expression in other human cancers, led us to hypothesise that these three genes, whose expression was restored by epigenetic drugs, could represent new potential drivers in cDLBCL.