Lastly, although SLC26A6 protein was reported to be present with SLC26A3 and A8 in sperm cells [76], its function in the mouse was shown to be dispensable for spermatogenesis and sperm fertilization potential [75] and, to date, no gene mutations in SLC26A6 have been shown to be associated with human male infertility. This evidence concerns the gene SLC26A3 and male infertility.