Further confirmations derived after a series of immunomodulant effects of PARPis were evidenced: the interaction with the tumor microenvironment (TME) of OC, the increased number of TILs, the upregulation of PD-L1, the enhanced antigen presentation and tumor mutational burden (TMB), and the interaction with the stimulator of interferon genes (STING) pathway [47,48,49,50,51,52]. Here, STING1 is linked to neoplasm.