Other authors observed that cytokine as IFN-γ and IL-33 levels were associated with OLP lesions, which also presented significant microbiota composition differences [89]; in fact, the bacterial community was less rich in erosive and reticular OLP patients than in healthy controls, with lower diversity in the erosive OLP group, and negatively correlated with salivary IL-17 concentration [90]. The gene discussed is IL33; the disease is oral lichen planus.