EGFR and neoplasm: Autophosphorylation inhibits angiogenesis, tumor cell proliferation, and programmed cell death; the PI3K/mTOR pathway governs immune resistance; inhibits angiogenesis, tumor cell proliferation, and death; suppresses Wnt1-MTA1-β-catenin signaling induced by leptin; STAT3 phosphorylation is reduced, and phospholipase D (PLD) activity is inhibited; induces cell cycle arrest and decreases mammosphere development, aldehyde dehydrogenases (ALDH) activity, and expression of iPSC inducers; EGFR is inhibited, and c-Src phosphorylation is suppressed