Encourages cell cycle arrest and death; prevents carcinogenesis, DNA damage, and cancer spread; Cells genetic and epigenetic profiles are altered, and COX activity is inhibited; NF-κB DNA’s binding activity is reduced, and cell viability, glucose ingesting, and ATP content are all reduced; TGFβ1 expression is suppressed; BCSC survival is reduced; Wnt/β-catenin signaling pathway is inhibited, resulting in autophagy; signaling between PI3K, Akt, and mTOR is suppressed. Here, AKT1 is linked to cancer.