Even more importantly, MEX [68], HAC [81] as well as AT [77] have been demonstrated to selectively suppress the growth of human acute myeloid leukaemia (AML) leukemic blasts from AML patients at concentrations (0.5, 0.5 and 0.05 μM, respectively) that did essentially not affect healthy donors’ CD34-positive myeloid progenitor cells. This evidence concerns the gene CD34 and acute myeloid leukemia.