SARS-CoV2 receptors, angiotensin-converting enzyme2 (ACE2), and transmembrane serine 2 protease (TMPRSS2), which may play a key role in intracellular invasion, are constitutively expressed and possibly upregulated on the brush border of the proximal tubule and in podocytes of the kidneys of COVID-19 patients [44]. The gene discussed is TMPRSS2; the disease is COVID-19.