Intriguingly, FMT from patients with higher SDI to naïve mice subjected to stroke procedures showed larger infarct volumes and severe neurological deficits, along with a higher number of proinflammatory (IL17+) γδ T cells and a lower number of (CD4+CD25+) T cells in the spleen than those of the corresponding mice after FMT from patients with a low SDI. This evidence concerns the gene IL17A and stroke disorder.