Similarly, the brain of GF mice subjected to distal MCAO showed doubled lesion volumes, impaired behavioral performance, and increased levels of inflammatory markers of Th1 and Th17 T cells (mRNA of IFN-γ and IL-17, respectively) after receiving dysbiotic post-stroke cecal contents when compared with mice receiving FMT from sham mice. The gene discussed is IL17A; the disease is Stroke.