TBK1 and vitiligo: Canonical pathway analysis showed the highest enriched pathway was ‘Autophagy’ (ATG2A, BNIP3, DAPK1, DAPK2, EIF2AK3, FOS, FOXO1, FOXO3, GABARAPL1, GABARAPL2, RGS19, RPTOR, and TBK1) and ‘Autophagy’ was predicted to be inhibited (z-score = −0.832) (Figure S1), which was consistent with our subsequent finding showing the lower ratio of LC3II/LC3I and higher p62 expression in vitiligo lesions.