EGFR and non-small cell lung carcinoma: In addition, miR-200c-3p and miR-203a-3p have been proposed as potential regulators of the resistance to epidermal growth factor receptor (EGFR) and tyrosine kinase inhibitors (TKIs) by modulating the epithelial-to-mesenchymal transition process through apoptosis in non-small-cell lung cancer (NSCLC) [41].