MTOR and idiopathic pulmonary fibrosis: Our KEGG pathway analysis of miR-200c-3p target genes showed MAPK and mTOR signaling pathways (Figure 6B), which have been studied, the p38 MAPK pathway has been studied in macrophages, Forkhead Box M1 (FOXM1) expression has been observed in IPF patients, in a mouse model deficient in FOXM1, an exacerbation of fibrosis was found, it was found that the deletion of this transcription factor activated the MAPK pathway, increasing fibrosis [42].