In order to do this, different mechanisms have been proposed: tumour growth can be caused through eosinophil-derived CCL22, by thymic stromal lymphopoietin (TSLP) that induces angiogenesis via VEGFA and by eosinophils-derived epidermal growth factor (EGF) along with TGF-β. The gene discussed is EGF; the disease is neoplasm.