Even with so many immune cells infiltrating the tumour, immune escape is made possible by mechanisms like an enhanced PD-L1 expression, WNT/β-catenin pathway (T cell infiltration inhibition), FasL expression (pro-apoptotic signal for tumour infiltrating lymphocytes or TILs) and androgen receptor erasure in Sertoli cells [110]. The gene discussed is AR; the disease is neoplasm.