Data obtained by our group demonstrated that the establishment of liver fibrosis by CCl4 in a murine model deregulated circadian clock by diminishing BMAL1, CLOCK, Per1, Per2, Per3, Cry1, Cry2, and retinoic acid receptor‐related orphan receptor (RORα) and increasing nuclear receptor subfamily 1 group D1 (REV‐ERBα) levels (González‐Fernández et al., 2018). The gene discussed is CLOCK; the disease is Hepatic fibrosis.