A variety of soluble proteins, including amyloid-β (Aβ), α-synuclein (α-syn), transthyretin (TTR), β2-microglobulin (β2-MG), and serum amyloid A (SAA) are aggregated and deposited in the body, causing amyloidosis (Picken, 2020). This evidence concerns the gene TTR and amyloidosis.