Based on our previous findings regarding the amygdala-related heightened baseline level of arousal even to nonthreatening stimuli as the neural mechanism underlying anxiety7, we hypothesized that MAOA-L and S allele carrying subjects would both exhibit distinct eMMN when compared to MAOA-H and non-carrier subjects, and that the formers’ eMMN would be equally associated with anxiety-related symptomatology. Here, MAOA is linked to Anxiety.