On the contrary, the pulmonary levels of pSer345‐MLKL, which is known to form homo‐/hetero‐oligomers mediating the plasma membrane rupture due to necroptosis,12, 38, 39, 40 were unchanged in PAH groups, despite its total form being upregulated in both disease stages. Here, MLKL is linked to pulmonary arterial hypertension.