Upregulation of CD69 in COVID-19 and non-COVID critical illness(61, 63), and correlation between the presence of MAIT cells and endotracheal CXCL10 and CXCL12 levels, showed these were highly activated unconventional T cells populating the airways of patients, suggesting a potential contribution in the regulation of local inflammation. The gene discussed is CD69; the disease is COVID-19.