SLC2A2 and glycogen storage disease due to GLUT2 deficiency: Mice lacking mTORC1 in RPTCs71 or treated with rapamycin develop glycosuria, phosphaturia, aminoaciduria, low-molecular weight proteinuria, and albuminuria72, findings observed in individuals suffering from the Fanconi-Bickel syndrome, caused by mutations in the GLUT2 gene, SLC2A273,74.