Interestingly, when we compared the lipid species associated with the single transgenics with lipid species associated with the double transgenics at the same time points, we found that at 1.5 and 3 dpt, Myc appeared to drive lipid species levels in the double transgenics, but that from 4.5 to 6 dpt, xmrk may supplant this role in x+m+ HCC liver samples (Table 1). This evidence concerns the gene MYC and hepatocellular carcinoma.