Importantly, we observed that the expression of HOXA1, HOXA2, and HOXA7 was distinctly increased in PCa specimens compared with nontumor specimens (Figures 3(b)–3(d)), while the contents of HOXA9 and HOXA10 were distinctly regulated in PCa samples in contrast to nontumor samples (Figures 3(e) and 3(f)). Here, HOXA9 is linked to posterior cortical atrophy.