C1QTNF9 and myocardial infarction: The downregulation of miR-214-3p results in elevation of CTRP9 expression, which in turn alleviates cardiac remodeling, including attenuated interstitial fibrosis, improved cardiac function, and enhanced survival rate in rat with MI chronic intermittent hypoxia, suggesting that CTRP9 may be a novel therapeutic target against pathologic remodeling (Du et al., 2020).