NEAT1 and myocardial infarction: Moreover, another study found that Neat1 was highly expressed in mice cardiomyocytes with ischemic cardiomyopathy/myocardial infarction, regulating the autophagy level through the control of Atg12 expression and related autophagy factors via targeting miR-378a-3p, which affected the proliferation, invasion, and metastasis of myocardial cells and alleviated hypoxia-induced myocardial cell injury (Zhao et al., 2020).