Interestingly, CFTRinh-172 induced a delay in wound repair, not only in non-CF cells (16HBE and corrected CFBE cells) but also in CFBE monolayers [165], indicating that this effect may not be related to the ion channel function of CFTR, but most probably is an unspecific effect of the inhibitor. Here, CFTR is linked to cystic fibrosis.