Huang et al. studied the expression of FcγRIIb, FcγRI, and FcγRIII on monocytes, T cells, and other cells in patients with BD and showed that FcγR is abnormally expressed in BD monocytes and is associated with disease progression and might promote the over-activation of monocytes in BD patients [53]. This evidence concerns the gene FCGR2A and Behcet disease.