In detail, our findings identified a state of stress with highly expressed heat shock proteins in Myo1, a highly vascular pericyte-like phenotype in Myo2, ACTG2+ myofibroblasts association in Myo3, antigen presentation phenotype in Myo4, and the characteristics of cancer-associated myofibroblasts in Myo5 [50]. This evidence concerns the gene ACTG2 and cancer.