The overexpression and/or mutation of EGFR is often found in GBs, which subsequently leads to the activation of many downstream signal pathways such as the phosphatidylinositol 3-kinase (PI3K)/AKT/mTOR pathway (Li et al., 2016), and this pathway is one of the almost inevitably altered molecular pathways in IDH-wild-type GB (Le Rhun et al., 2019). The gene discussed is MTOR; the disease is Guillain-Barre syndrome.