Recently, Huang et al. reported that interleukins (IL-1β, IL-7, IL-8, IL-9, and IL-10), fibroblast growth factor (FGF), tumor necrosis factor-α (TNF-α), granulocyte colony-stimulating factor (G-CSF), interferon (IFN), monocyte chemoattractant protein (MCP1), and macrophage inflammatory protein 1-α (MIP1-α) were significantly upregulated in the COVID-19 patients [34] An increase in von Willebrand factor antigen, von Willebrand factor activity, and factor VIII levels also contributes to the atherosclerotic plaque. This evidence concerns the gene TNF and COVID-19.