used microarrays to compare LNCRNA and mRNA expression in keratin-forming cells from patients with psoriasis and healthy patients, showing that the miRNA maximally linked to LNCRNA and mRNA was miR-484, adding some theoretical basis for miR-484 in the mechanism of psoriasis (53) (Table 2). Notably, the ability of miR-484 to negatively regulate the TNF signaling pathway in cervical carcinogenesis development was previously reported (32). The gene discussed is TNF; the disease is psoriasis.