Whole ventricle analysis showed that phosphorylation of MST1, LATS1, and YAP were higher in MI than in sham‐operated mice, consistent with our in vitro finding that MST1 and LATS1 were activated after MI, along with elevated phosphorylation and subsequent nuclear exclusion of YAP and downregulation of CTGF and CYR61. This evidence concerns the gene LATS1 and myocardial infarction.