However, several pathogenic theories have been proposed: in the early stages of AP, the release of serum catecholamines and glucagon, as well as pancreatic lipases, in response to stress accelerates the decomposition of the adipose tissue with the consequent release of triglycerides as its degradation products (free fatty acids, FFA) through the systemic circulation [35]. Here, PNLIP is linked to alkaline phosphatase measurement.