Fusion proteins known to act as leukemia-driving lesions or signatures were enriched in our proteomics dataset, including PBX1, ROR1, and WNT16 for the TCF3-PBX1 fusion26, and ABL1 for BCR-ABL1, NUP214-ABL1, and SPFQ-ABL1 fusions (Fig. 1d). The gene discussed is ABL1; the disease is leukemia.