RAX and Nematoda infectious disease: Although the rax1 mutant displays lower ROS accumulation under nonstress conditions, and CNs can utilize ROS for successful parasitism, the mutant's overproduction of ROS on nematode infection (caused by the misregulation of the ascorbate–glutathione cycle, resulting in oxidative stress and enhanced camalexin production) might hamper nematode development (Ball et al., 2004; Noctor & Foyer, 1998; Siddique et al., 2014; Tierens et al., 2002; Zhao et al., 1998).