SOAT1 and leukemia: The mutational profiles of pediatric leukemia have also been studied in different ethnic groups revealing recurrent mutational hotspots, driver genes and variants involved in different pathways: RTK/RAS signaling and its downstream MAPK/ERK signaling, PI3K/AKT and MTOR, JAK/STAT signaling, Notch signaling, WNT/β-catenin, CXCL12, NF-κB, Metabolic and other pathways, including p53 [11–14].