In addition to lipogenesis and adipogenesis, METTL3 was found to regulate glucose metabolism in colorectal cancer, as METTL3-mediated m6A promoted the mRNA stability of Hk2 and Glut1 in an m6A and IGF2BP2/3-dependent manner (Shen et al., 2020). This evidence concerns the gene SLC2A1 and colorectal cancer.