Interestingly, METTL3 expression in ovarian cancer also increased migration and induction of EMT through increased protein expression of the receptor tyrosine kinase AXL; however, while the Axl mRNA transcript contains fourteen m6A sites, METTL3 regulation of AXL translation is believed to be m6A-independent (Hua et al., 2018). This evidence concerns the gene METTL3 and ovarian carcinoma.