This study also demonstrated the role of miR‐29a, miR‐9, miR‐30d, miR‐124a, miR‐146a, miR‐34a, and miR‐375 in fine‐tuning insulin secretion and pathogenesis of T2DM, while their expression levels remained steady in the pre‐T2DM stage, refusing their potential relevance as disease‐specific markers.14 The gene discussed is INS; the disease is type 2 diabetes mellitus.