INS and type 2 diabetes mellitus: According to the aforementioned evidence, type 2 diabetes and NODAT seem to share the following phenomena: A) a diminished secretory capacity of β cells, B) a decrease in glucose-induced insulin secretion, C) an increase in the apoptosis of pancreatic islets, and D) the deterioration of the metabolic pathways involved in the synthesis of ATP and the nuclear factor of activated T-cells (NFAT).