The inhibition of constitutive NF-κB activation can curb tumor growth and metastasis and sensitize pancreatic tumor cells to the apoptosis induced by the anti-cancer agent.19 Its role in malignant transformation, differentiation, cell cycle, and apoptosis have made NF-κB an interesting target for clinical intervention.20 Interestingly, several miRNAs, including miR-155,21,22 miR-223,23 miR-222,24 miR-21,25 and miR-1908,26 have been reported to contain an NF-κB site through which NF-κB transcriptionally regulates their expression. Here, NFKB1 is linked to pancreatic neoplasm.