CD4 and systemic lupus erythematosus: Given the aforementioned results that TFH‐AIM2 may have a function in SLE patients and deficiency of CD4+ T‐cell‐intrinsic Aim2 downregulates TFH cell differentiation, we suspected that Aim2 deficiency could impair TFH cell differentiation, which alleviated clinical lupus symptoms after pristane administration.