Cervical carcinomas develop an abnormal TME characterized by interstitial hypertension, intratumoral hypoxia, elevated amounts of lactate, nitric oxide, arginase, vascular endothelial growth factor (VEGF), reactive oxygen (ROS), low glucose concentrations, and energy deprivation, resulting in tumor neovascularization [11, 12]. Here, VEGFA is linked to cervical carcinoma.