DLGAP1-AS1 sequestered miR-486-5p to promote cell proliferation of hepatocellular cancer [20], and DLGAP1-AS1 urged the occurrence of liver cancer and epithelial-mesenchymal transition through the feedback loop of the miR-26a/b-5p/IL-6/JAK2/STAT3 and Wnt/β-catenin pathways [21]. The gene discussed is STAT3; the disease is hepatocellular carcinoma.