MOPV infection activated the following pathways relative to the mock condition: chemokine signaling, oxidative phosphorylation, purine metabolism, cell-adhesion molecules, pentose phosphate, complement-coagulation cascade (at 24 h for the last three), RIG-I signaling receptor (Figure 2C), TGFβ signaling, and IFNα signaling (at 6 and 24 h) (Figure 2D). Here, TGFB1 is linked to infection.