In this research, BCP induced apoptosis of H22 tumor cells mainly through the third pathway, the cell cycle analysis, FITC-AnnexinV/PI staining, and JC-1 experiments, which have collectively shown that BCP is sufficient to induce apoptosis and inhibit the rapid proliferation of H22 hepatocarcinoma cells in a dose-dependent manner. Here, ANXA5 is linked to neoplasm.