At the genetic level, CM normalizes the histopathology of patients with diabetes, affects vital enzymes and proteins for cardiovascular and hepatorenal functions, controls phosphoenolpyruvate carboxykinase (PEPCK) gene transfer that induces a gluconeogenesis effect, and plays a pivotal role in regulating a set of metabolizing enzymes that affect carbohydrates and lipids metabolism [58]. The gene discussed is PCK2; the disease is diabetes mellitus.